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+86 199 3313 8870
Mar. 02, 2021
Chicken glandular gastritis is an infectious disease characterized by glandular stomach enlargement and muscular gastric erosion. The clinical manifestations of sick chickens are depression, reduced feed intake, growth retardation, weight loss, and poor orderliness of the flock. The disease is currently on the rise in the large-scale chicken breeding industry, which has caused huge economic losses to the breeding industry.
Gastroenteritis widely occurs in chicken flocks of various breeds, including broilers, laying hens, breeders, etc., mainly in commercial broiler chickens, and the occurrence of this disease is not seasonal, and it occurs throughout the year. Especially in the hot summer and autumn, the incidence is higher. At present, the incidence area of the disease is expanding, and the disease can be seen in all parts of the country. The disease occurs in a wide range of ages, and can be observed in 1-day-old chicks. The disease can also occur in 300-day-old layer breeders, but it mostly occurs in 7-30-day-old commercial broilers. The age of chicken onset is also closely related to the severity of the disease. The younger the onset age, the higher the incidence, and the more severe the symptoms. For chickens with onset of 1 day of age, growth inhibition can be seen at 3 days of age. Infected chickens at older ages have a relatively lower incidence, but they also show symptoms of weight loss and disheveled feathers; adult chickens are less morbid than chicks. Eggs and breeders generally exhibit recessive infections, which are manifested by decreased feed intake. The egg production rate has dropped. When the disease occurs alone, the death rate is lower, and when other diseases occur, the death rate increases.
Sick chickens and infected chickens are the main source of infection. The disease is mainly infected through the digestive tract through contaminated chicken houses, feed and drinking water. Once the disease occurs in an area or chicken farm, it is difficult to completely eliminate it. The disease can be transmitted horizontally and vertically, and clinically, it is mainly horizontal. The morbidity and mortality of chicken flocks are closely related to the conditions of feeding and management. The morbidity and mortality of the well-managed chickens are relatively light, and the mortality and morbidity of the poorly managed chickens are more serious.
Diseased chickens are characterized by rough and dull feathers, pale crests and beards, atrophy, pale mucous membranes, and dry skin on the legs; sick chickens have decreased appetite, reduced feed conversion rate, undigested feed in the feces, and faeces are yellow and soft. , Showing oily "fish intestine-like" or rotten carrot-like, individual discharge of brownish red to black loose stools; some sick chickens have fluid accumulation in the crops, and the neck is enlarged; some chickens have tears, swollen eyes, head shaking and respiratory tract Symptoms, snoring can be heard in large groups.
In the middle and late stages of the disease, the respiratory symptoms of the chickens basically disappeared. The sick chickens are depressed, stand up with closed eyes, droopy wings, disheveled feathers, and drastically reduce feed intake and water consumption; individual diseased corns are cloudy and unclear conjunctiva, and some are affected by blindness Feed. Sick chickens become thinner gradually, their production level drops, chicken growth is slow or stagnant, resulting in serious unevenness of large groups, weight is not up to standard, and the weight is only 1/3-1/2 of healthy chickens, showing a negative growth trend, and eventually exhaustion And die.
The main lesions of necropsy were in the glandular stomach and muscular stomach. The glandular stomach is swollen and milky white, the glandular papilla is swollen and bleeding, the glandular gastric mucosa is swollen and thickened, the nipple boundary is fused, the glandular stomach wall is thickened and edema, and the milky liquid can be squeezed gently with the fingers; the muscular gastric endomembrane Bleeding, thickening, hardening and cracking, severe crater-like ulcers and even gastric perforation. Immune organs such as thymus, spleen and bursa of Fabricius are severely atrophy; the intestinal wall becomes thin and bleeding, the intestinal mucosa falls off and is filled with undigested feed; the gallbladder is filled with dark green, bile overflows, the crop is expanded, and there is dark brown mucus inside.
Gastrointestinal gastritis in chickens is a comprehensive disease caused by one or several factors. There is no fixed specific medicine for treatment. Therefore, in actual production, prevention is the first to improve the body's immunity.
In normal times, it is necessary to strengthen feeding management, strict disinfection, improve environmental sanitation, strengthen sanitation and disinfection of chickens, reduce feeding density, ensure temperature, and reasonable ventilation. Adjust the nutritional balance of feed, pay attention to the quality of animal protein and oil, adjust the crude fiber content of feed, increase the content of protein and vitamins, etc. At the same time, monitor the quality of raw materials, prevent the occurrence of mycosis, and timely add mold to the feed. It can prevent mycotoxin poisoning and eliminate the inducement of feed mildew and malnutrition. Increase the number of feeds to reduce the residence time of the feed in the chicken house to ensure the freshness of the feed and prevent the feed from spoiling. The shorter the feed stays in the chicken house, the lower the incidence of the glandular stomach.
Strictly control and detect the epidemic diseases transmitted vertically by breeder flocks, and purify important vertical infectious diseases such as chicken reticuloendothelial hyperplasia for breeder flocks. Select breeder farms with good breeding and management to introduce chicks to strengthen the control of diseases such as Newcastle disease and infectious bronchitis.
Once adenomyosis gastritis is found, detailed differential diagnosis should be carried out, the cause of the disease should be analyzed, the pathogenic factors should be eliminated, and the symptomatic treatment should be carried out. On the basis of the treatment, adjuvant treatment can be carried out to promote the recovery of the body and improve the production performance. First of all, it is necessary to repair the ulcer of the glandular stomach and restore the secretory function of the glandular stomach. Secondly, it is necessary to supplement the body's vitamins, which can promote gastrointestinal peristalsis, enhance liver detoxification, promote digestive gland secretion, increase feed conversion rate, and quickly eliminate feed stool. Third, control secondary infections and treat gastrointestinal diseases. Finally, it activates gastrointestinal function, activates glandular stomach epithelial cells, and improves feed intake and disease resistance.
Treatment plan-adenomyosis dual effect
